Medical science has claimed for decades that elevated cholesterol levels pose a health threat, especially if you have too much of the so-called “bad” cholesterol – or LDL (Low-Density Lipoprotein). However, a team of international experts is now arguing that this theory is exaggerated. In fact, the experts specifically advise against taking cholesterol-lowering statins. Meanwhile, science is focused on how to improve the safety of statins by combining them with supplements of coenzyme Q10.
There is absolutely no reason not to consume eggs, meat, butter and other cholesterol-filled foods with a good conscience. American dietary guidelines have finally exonerated cholesterol, which happens to be an essential compound. Many scientists actually claim that atherosclerosis and cardiovascular disease are both a result of inflammation and lack of specific micronutrients. It is important to pay attention to factors that are known to promote inflammation in the body and take the necessary steps by looking after your circulatory system, making healthy lifestyle choices, and possibly even using supplements.
Eat plenty of oily fish from clean oceans or take a fish oil supplement to make sure that you get enough of the essential omega-3 fatty acids. It lowers your risk of premature death by protecting you against atherosclerosis and a number of other diseases. In fact, blood levels of omega-3 tell a lot more than levels of cholesterol, according to a new study that is published in Journal of Clinical Lipidology.
- but ageing processes and cholesterol-lowering drugs inhibit the body’s Q10 synthesis
No matter how you twist and turn it, cholesterol is an essential substance, and we humans produce most of it ourselves. What is important is to make sure that the cholesterol we have in our blood does not oxidize, and that is something which Q10 and other antioxidants take care of. Stable blood sugar levels also help us maintain a healthy cholesterol balance.
Cholesterol-lowering statins are among some of the most widely sold medical drugs. However, there is growing disagreement about their preventive effect on atherosclerosis and cardiovascular diseases, which are linked to many other factors than cholesterol levels alone. It turns out that statins also block the body’s production of vitamin K2, a nutrient that contributes to removing calcium from the bloodstream. A new study that is published in Medical Sciences concludes that atherosclerosis is more prevalent among statin users than among non-users, regardless of any cardiovascular diseases that have already been diagnosed. In other words, statins actually increase your risk of clogged-up arteries and that contradicts the traditional view. The new study supports earlier research, and it has been known for years that statins also block the endogenous synthesis of coenzyme Q10 and vitamin D, both of which are important for the heart and circulatory system.
A growing number of people suffer from overweight and type 2 diabetes, both of which are problems that come at a cost both to society and to the individual. The traditional dietary guidelines are not of much use, and many people find themselves in a hopeless battle because they also suffer from insulin resistance with impaired cellular glucose uptake. Countless epidemiological studies have demonstrated that the widespread lack of vitamin D contributes to the problem. In a review article that is published in Nutrients, the scientists address vitamin D’s many functions with relation to blood glucose regulation, satiety, body weight, and prevention of type 2 diabetes. It should be noted that overweight people and type 2 diabetics may have an increased need for vitamin D, and magnesium is also required for activating the vitamin.
Pantothenic acid originates from the Greek word pantos, which means everywhere. As the word suggests, this vitamins is found in both plants and animals. Vitamin B5 is water-soluble and because it does not get stored in the body we depend on regular intake of the nutrient. Vitamin B5 is destroyed by heating and freezing.