Alzheimer’s is an insidious disease and the leading cause of dementia. It’s also one of the major causes of death in old age. Diet plays a major role in preventing the disease. In fact, having high concentrations of the omega-3 fatty acid, DHA, in the blood can halve the risk of developing Alzheimer’s disease, according to a study that is published in Nutrients. DHA (docosahexaenoic acid) is found in oily fish and fish oil supplements. This essential fatty acid is also found in all our cell membranes (including nerve cells) and plays a key role in maintaining our general health and cognitive skills. Unfortunately, modern diets contain far too little omega-3, but science is not quite sure how much we need.

Everyone is affected by electromagnetic radiation from cell phones, device screens, the electricity supply and other electric systems. The brain’s neurons are particularly vulnerable, especially because electromagnetic radiation can increase the neuronal uptake of calcium ions, which makes the neurons overactive and may even cause them to perish. A study published in News Medical Life Sciences shows that too much calcium in the brain increases the risk of Alzheimer’s disease and digital dementia in young individuals. The increased electromagnetic radiation also generates loads of free radicals in the body that can cause oxidative stress and cellular damage because it outperforms the body’s different antioxidants. This was described in an article published in The Journal of Microscopy & Ultrastructure that also mentions how electromagnetic radiation can cause stress, headaches, fatigue, anxiety, and other symptoms.

Neurological disorders such as Alzheimer’s disease, dementia, depression, Parkinson’s disease, stroke, migraine, and fibromyalgia are rather common. These conditions are a result of imbalances in the nervous system, and they are often insidious. In a new review article that is published in Frontiers in Neuroscience, researchers have studied Q10’s role in the different neurological disorders. The reason for this is that Q10 is important for cellular energy turnover and serves as an antioxidant that protects the circulatory system and the nerve cells against oxidative damage. As we grow older, our endogenous Q10 synthesis decreases, and certain diseases and medical drugs also impair the body’s Q10 synthesis.

The brain is particularly vulnerable towards oxidative stress and local inflammation that can set the stage for Alzheimer’s disease and other neurological conditions. However, it turns out that certain selenium-containing antioxidants are able to protect the brain neurons against damage. Also, selenium supplements can improve cognitive performance in patients suffering from mild cognitive impairment and Alzheimer’s disease, according to a meta-analysis that is published in Nutrients.

The number of seniors worldwide is increasing and more and more are affected by Alzheimer’s disease, the leading cause of dementia. We should therefore focus much more on this health problem that comes with an enormous human and socio-economic price tag. According to a new meta-analysis, supplementation with B vitamins appears to prevent or delay mild cognitive impairment in older people, which is normally an early sign of Alzheimer’s disease. In fact, supplementation with B vitamins may even represent an inexpensive therapy form with a huge potential.

- and other types of organ damage

Vitamin K occurs naturally in different forms. The vitamin is primarily known for its role in blood coagulation, but a team of German scientists has found a new type of vitamin K. This form serves as a very particular type of antioxidant that counteracts cell death caused by a process called ferroptosis. Because ferroptosis is involved in the development of Alzheimer’s disease, acute organ damage, and other diseases, the researchers see a whole new potential for vitamin K in the prevention and mitigation of ferroptosis-related ailments. Earlier studies even suggest that selenium-containing antioxidants also protect against cell death caused by ferroptosis.